Lambert-Eaton-Myasthenie-Syndrom (LEMS): Pathophysiologie und Behandlung
Über den Vortrag
Der Vortrag „Lambert-Eaton-Myasthenie-Syndrom (LEMS): Pathophysiologie und Behandlung“ von Roy Strowd, MD ist Bestandteil des Kurses „Störungen der neuromuskulären Verbindungsstelle“.
Quiz zum Vortrag
Which of the following is not consistent with the pathophysiology of Lambert-Eaton myasthenic syndrome?
- Slow, repetitive nerve stimulation results in increased response due to a calcium build-up in the presynaptic terminus.
- Voltage-gated calcium channel antibodies prevent calcium influx into the presynaptic terminus.
- Reduced acetylcholine (ACh) release results in a decrease in baseline motor amplitude.
- Slow, repetitive nerve stimulation results in diminished response due to a reduction in ACh release.
- Fast repetitive nerve stimulation increases the buildup of calcium in the presynaptic terminus resulting in an incremental response.
Which of the following is not a treatment for Lambert-Eaton myasthenic syndrome?
- Pyridostigmine
- Corticosteroids
- 3,4-diaminopyridine
- Plasmapheresis
- Immunosuppression
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Dozent des Vortrages Lambert-Eaton-Myasthenie-Syndrom (LEMS): Pathophysiologie und Behandlung
Roy Strowd, MD
Dr. Roy Strowd is the Assistant Dean of Undergraduate Medical Education at the Wake Forest Baptist Medical Center, in North Carolina, USA.
He obtained his MD from Wake Forest School of Medicine in 2009, and his MEd from Johns Hopkins University in 2021. Currently, he co-directs the Neurofibromatosis and Tuberous Sclerosis Clinics at Wake Forest Baptist Medical Center, and is a researcher for the Adult Brain Tumor Consortium (ABTC) and Alliance for Clinical Trials in Neuro-Oncology.
Due to his achievements, he earned the M. Brownell Anderson Award from the Association of American Medical Colleges.
Within Lecturio, Dr. Strowd teaches courses on Clinical Neurology.
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