Botulinumtoxin-Vergiftung: Pathophysiologie und Behandlung
Über den Vortrag
Der Vortrag „Botulinumtoxin-Vergiftung: Pathophysiologie und Behandlung“ von Roy Strowd, MD ist Bestandteil des Kurses „Störungen der neuromuskulären Verbindungsstelle“.
Quiz zum Vortrag
Which of the following best characterizes the mechanism of botulism toxicity?
- Botulinum toxin acts as a protease and cleaves SNARE proteins which are required for the binding of the presynaptic vesicle to the nerve membrane.
- Botulinum toxin acts as a phospholipase that degrades the vesicle containing the neurotransmitter.
- Botulinum toxin binds to ACh receptors and acts as a competitive inhibitor preventing muscle contraction.
- Botulinum toxin blocks presynaptic calcium channels preventing synaptic depolarization.
- Botulinum toxin augments the reuptake of ACh in the synaptic cleft resulting in decremental response with repetitive action.
Which of the following is not a treatment of iatrogenic botulism?
- Broad-spectrum antibiotic therapy
- Withdrawal of offending agent or ingestion
- Antitoxin
- Pyridostigmine
- Supportive care
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Dozent des Vortrages Botulinumtoxin-Vergiftung: Pathophysiologie und Behandlung
Roy Strowd, MD
Dr. Roy Strowd is the Assistant Dean of Undergraduate Medical Education at the Wake Forest Baptist Medical Center, in North Carolina, USA.
He obtained his MD from Wake Forest School of Medicine in 2009, and his MEd from Johns Hopkins University in 2021. Currently, he co-directs the Neurofibromatosis and Tuberous Sclerosis Clinics at Wake Forest Baptist Medical Center, and is a researcher for the Adult Brain Tumor Consortium (ABTC) and Alliance for Clinical Trials in Neuro-Oncology.
Due to his achievements, he earned the M. Brownell Anderson Award from the Association of American Medical Colleges.
Within Lecturio, Dr. Strowd teaches courses on Clinical Neurology.
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