Stroke: Pathophysiology
Über den Vortrag
Der Vortrag „Stroke: Pathophysiology“ von Roy Strowd, MD ist Bestandteil des Kurses „Stroke and Intracranial Hemorrhage“.
Quiz zum Vortrag
What is the normal rate of cerebral blood flow?
- Normal CBF is > 50 ml per 100 g brain tissue per minute.
- Normal CBF is > 10 ml per 100 g brain tissue per minute.
- Normal CBF is > 30 ml per 100 g brain tissue per minute.
- Normal CBF is > 75 ml per 100 g brain tissue per minute.
- Normal CBF is > 100 ml per 100 g brain tissue per minute.
What is described by the term "penumbral tissue?"
- An area of ischemic tissue surrounding the tissue affected by a stroke that is still potentially salvageable
- An area of normally perfused brain tissue
- An area of non-functioning brain tissue that has suffered infarction due to a stroke
- An area of the brain that has previously been affected by a stroke
- An area of brain tissue that has not been affected by a recent stroke
What is a very serious complication due to brain edema and mass effect after a stroke?
- Brain herniation
- Sodium/potassium channel dysfunction
- Penumbral tissue transformation
- Venous sinus thrombosis
- Thalamic infarction
How long after a stroke is the greatest risk of hemorrhagic transformation due to reperfusion?
- Hours to days after the event
- Days to weeks after the event
- Weeks to months after the event
- Months to years after the event
- Seconds to minutes after the event
What is meant by the phrase "time is brain?"
- Acute intervention during a time of cerebral ischemia can prevent a brain infarction and its associated clinical manifestations.
- Acute intervention after a brain infarction can prevent the extension of the stroke.
- Prevention of a stroke by risk factor modification saves brain tissue.
- Acute intervention after a brain infarction can prevent hemorrhagic transformation.
- Human brain tissue is rapidly and irretrievably lost as a stroke progresses and therapeutic intervention cannot prevent deterioration.
After acute occlusion of a cerebral blood vessel, what is the next step in the cascade of events?
- Sodium/potassium channels in the cells become dysfunctional
- Water influx into brain cells causes edema
- Brain swelling is seen on CT or MRI imaging
- Blood vessels become dysfunctional causing vasogenic edema and apoptosis
- Infarction and necrosis of brain tissue result
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Dozent des Vortrages Stroke: Pathophysiology
Roy Strowd, MD
Dr. Roy Strowd is the Assistant Dean of Undergraduate Medical Education at the Wake Forest Baptist Medical Center, in North Carolina, USA.
He obtained his MD from Wake Forest School of Medicine in 2009, and his MEd from Johns Hopkins University in 2021. Currently, he co-directs the Neurofibromatosis and Tuberous Sclerosis Clinics at Wake Forest Baptist Medical Center, and is a researcher for the Adult Brain Tumor Consortium (ABTC) and Alliance for Clinical Trials in Neuro-Oncology.
Due to his achievements, he earned the M. Brownell Anderson Award from the Association of American Medical Colleges.
Within Lecturio, Dr. Strowd teaches courses on Clinical Neurology.
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I loved how he took his time breaking the pathophysiology of the ischemic stroke in a way that I don't need to look up any terminology to understand him better
